Regulation of heat-loss responses during steady-load exercise with muscle ischemia or hypoxemia has been reported to be modulated according to changes in relative work rate and body exertion. The present study demonstrated that increase in perception of exertion and relative oxygen uptake (%VO2max) induced by progressive increase in muscle fatigue has no influence on exercise temperature regulation. The link between perceived exertion and perturbation in thermoregulatory responses observed during exercise with reduced oxygen delivery must therefore be of peripheral origin.
COBISS.SI-ID: 3178859
The results of postdoctoral project on ischemic training are presented in the article. Based on the non-invasive real-time oxygen kinetics measurements (NIRS) and surface EMG amplitudes it is demonstrated that a 50% increase in endurance capacity of m. quadriceps observed with ischemic training can be attributed to enhanced muscle blood supply and oxygen availability rather then to increased muscle activation. The small increase in muscle cross-sectional area may thus be an evidence of ischemia-induced capillarisation and increased muscle glycogen stores rather than true muscle hypertrophy.
The results of postdoctoral project on hypoxic training are presented in the article. The effect of acute and chronic intermittent exposures to normobaric hypoxia (FO2= 11%) during exercise on muscle cross-sectional area and performance are reported. In addition, their influence on cardiovascular responses, muscle activation and oxygen kinetics is evaluated. The key findings is that hypoxic training does not have an augmented influence on observed parameters compared to normoxic training conditions, despite a modest reduction in muscle oxygen availability during acute hypoxic exposures.